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In ferroptosis, lysosomal iron release catalyzes lipid peroxidation, overwhelming antioxidant defenses (82), while lysosomal impairment compromises mitochondrial health, potentially precipitating ferroptosis (83)

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The same study also reported that fluoxetine caused an increase in antioxidant enzyme capacity, as measured by increased catalase and glutathione-S-transferase activities in rat hippocampus [91]